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Language disorders sit at the intersection of some of psychology's most important domains: brain localization, developmental psychology, learning processes, and social behavior. When you study these disorders, you're not just memorizing symptoms—you're learning how damage to specific brain regions produces predictable deficits, how developmental timelines affect language acquisition, and how communication difficulties ripple outward into anxiety, social isolation, and academic struggles. The AP exam loves to test your understanding of brain-behavior relationships, particularly through aphasia questions that ask you to connect lesion location to specific symptoms.
Don't approach this list as ten separate disorders to memorize. Instead, focus on the underlying mechanisms: acquired vs. developmental onset, expressive vs. receptive deficits, neurological vs. psychological causes. When you understand why Broca's aphasia produces halting speech while Wernicke's aphasia produces fluent nonsense, you can reason through any question the exam throws at you. Know what concept each disorder illustrates, and you'll be ready for both multiple choice and FRQ applications.
These disorders result from injury to a previously healthy brain, typically from stroke, trauma, or disease. The specific symptoms depend on which brain region is damaged, making these disorders powerful evidence for localization of function—a concept the AP exam tests repeatedly.
Compare: Broca's Aphasia vs. Wernicke's Aphasia—both result from left hemisphere damage, but Broca's impairs production while Wernicke's impairs comprehension. If an FRQ asks about localization of function, these two disorders are your strongest examples of how different brain regions control different aspects of the same cognitive ability.
These disorders emerge during childhood development rather than from injury to a mature brain. The underlying causes involve atypical neural development or genetic factors, and early intervention is critical because the brain's plasticity decreases with age.
Compare: SLI vs. DLD—these terms are often used interchangeably, but DLD is the newer, broader diagnostic category. Both emphasize that language difficulties can occur in otherwise typically developing children, challenging the assumption that language problems always signal general cognitive impairment.
These learning disabilities specifically target the processing of written language. They involve difficulties with the visual-phonological connections required for decoding text or the motor-cognitive coordination needed for writing.
Compare: Dyslexia vs. Dysgraphia—dyslexia impairs reading (language input), while dysgraphia impairs writing (language output). Both are specific learning disabilities that occur despite normal intelligence, demonstrating that academic skills can be selectively impaired.
These disorders affect how smoothly speech flows or how effectively individuals navigate social communication contexts. The underlying mechanisms range from motor control issues to anxiety to broader neurodevelopmental differences.
Compare: Stuttering vs. Selective Mutism—stuttering involves difficulty with speech production across contexts, while selective mutism involves intact speech ability that anxiety blocks in specific situations. Both demonstrate how psychological factors (anxiety, stress) interact with communication, but their underlying mechanisms differ completely.
These broader developmental conditions include communication difficulties as one component of a larger syndrome. Language impairments in these disorders reflect differences in how the brain processes social and communicative information.
Compare: ASD vs. Specific Language Impairment—both can involve language delays, but ASD includes social interaction differences and restricted interests that SLI does not. This distinction matters for understanding whether language difficulties are isolated or part of a broader developmental pattern.
| Concept | Best Examples |
|---|---|
| Localization of function | Broca's Aphasia, Wernicke's Aphasia |
| Acquired vs. developmental onset | Aphasia (acquired) vs. DLD (developmental) |
| Expressive language deficits | Broca's Aphasia, Dysgraphia, Stuttering |
| Receptive language deficits | Wernicke's Aphasia |
| Specific learning disabilities | Dyslexia, Dysgraphia |
| Anxiety-related communication | Selective Mutism, Stuttering (secondary symptoms) |
| Language without other cognitive impairment | SLI, DLD, Dyslexia |
| Social communication difficulties | ASD, Selective Mutism |
Both Broca's aphasia and Wernicke's aphasia result from left hemisphere damage. What specific difference in lesion location explains why one produces non-fluent speech while the other produces fluent but meaningless speech?
A child speaks fluently at home but has never spoken a word at school despite two years of attendance. Which disorder does this describe, and why is it classified as an anxiety disorder rather than a language disorder?
Compare and contrast dyslexia and dysgraphia: What do they share as specific learning disabilities, and how do their core deficits differ in terms of language input versus output?
If an FRQ asks you to explain how brain damage can produce different language deficits depending on location, which two disorders provide the strongest contrasting examples, and what would you emphasize about each?
A student has significant vocabulary and grammar difficulties but scores normally on nonverbal IQ tests and has no hearing impairment. Which developmental language disorder does this pattern suggest, and what does it reveal about the modularity of language in the brain?