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🫦Intro to Human Sexuality

Common Sexual Dysfunctions

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Why This Matters

Sexual dysfunctions represent a critical intersection of biological, psychological, and social factors—the biopsychosocial model you'll see throughout human sexuality coursework. These conditions aren't just medical issues; they reflect how arousal, desire, orgasm, and pain responses interact with relationship dynamics, cultural expectations, and mental health. Understanding dysfunctions helps you grasp how the sexual response cycle works when it's functioning well, and what happens when any phase gets disrupted.

You're being tested on your ability to distinguish between desire-phase disorders, arousal-phase disorders, and orgasm-phase disorders, as well as recognizing when pain or aversion creates barriers to sexual functioning. Don't just memorize disorder names—know which phase of the sexual response cycle each dysfunction affects, whether the primary cause is typically physiological or psychological, and how treatment approaches differ based on underlying mechanisms.


Desire-Phase Dysfunctions

These disorders occur at the very beginning of the sexual response cycle, affecting motivation and interest before physical arousal even begins. The desire phase involves cognitive and emotional components—sexual thoughts, fantasies, and receptivity to sexual cues.

Female Sexual Interest/Arousal Disorder

  • Reduced or absent sexual interest—characterized by diminished sexual thoughts, fantasies, and responsiveness to erotic cues
  • Combines desire and arousal criteria in DSM-5, recognizing that these phases often overlap in women's sexual experiences
  • Multifactorial etiology including hormonal fluctuations, relationship satisfaction, and psychological stressors like depression or body image concerns

Male Hypoactive Sexual Desire Disorder

  • Persistently deficient sexual desire causing clinically significant distress, distinct from asexuality or normal variation
  • Must rule out medical causes—low testosterone, medications, or other conditions before diagnosis applies
  • Psychological contributors include chronic stress, relationship conflict, and depression; treatment often combines therapy with lifestyle modifications

Sexual Aversion Disorder

  • Extreme avoidance of genital contact—goes beyond low desire to active fear, disgust, or anxiety about sexual activity
  • Trauma-linked etiology often involves past sexual abuse, assault, or intensely negative early sexual experiences
  • Treatment focuses on underlying causes through trauma-informed therapy, gradual desensitization, and addressing anxiety responses

Compare: Male Hypoactive Sexual Desire Disorder vs. Sexual Aversion Disorder—both involve avoiding sex, but hypoactive desire reflects lack of motivation while aversion reflects active avoidance due to fear or disgust. If an essay asks about trauma's impact on sexuality, aversion disorder is your clearest example.


Arousal-Phase Dysfunctions

Arousal disorders involve difficulty achieving or maintaining the physiological changes necessary for sexual activity, even when desire is present. The arousal phase depends on vasocongestion (blood flow to genitals) and myotonia (muscle tension), both regulated by the autonomic nervous system.

Erectile Dysfunction (ED)

  • Inability to achieve or maintain erection sufficient for satisfactory sexual performance; the most commonly discussed male sexual dysfunction
  • Vascular and neurological causes predominate—diabetes, cardiovascular disease, and nerve damage disrupt blood flow mechanisms
  • Psychological factors create feedback loops—performance anxiety after one episode can perpetuate the problem; treatment includes PDE5 inhibitors, therapy, and addressing underlying health conditions

Persistent Genital Arousal Disorder (PGAD)

  • Unwanted, intrusive genital arousal occurring without sexual desire or stimulation—essentially the opposite of typical arousal disorders
  • Causes significant distress and functional impairment; arousal may persist for hours or days without relief from orgasm
  • Poorly understood etiology potentially involving neurological abnormalities, pelvic nerve dysfunction, or medication side effects; treatment options remain limited

Compare: Erectile Dysfunction vs. PGAD—ED involves insufficient physiological arousal response, while PGAD involves excessive and unwanted arousal. Both demonstrate how arousal mechanisms can malfunction in opposite directions.


Orgasm-Phase Dysfunctions

These disorders affect the climax phase of sexual response, either through timing problems or inability to reach orgasm despite adequate stimulation. Orgasm involves rhythmic contractions of pelvic muscles and is mediated by both sympathetic nervous system activation and learned response patterns.

Premature Ejaculation

  • Ejaculation occurring within approximately one minute of penetration, before the person wishes—the most common male sexual dysfunction
  • Serotonin regulation plays a key role—low serotonin activity in ejaculatory control centers contributes to rapid response
  • Behavioral treatments effective—stop-start technique and squeeze method help build ejaculatory control; SSRIs sometimes prescribed off-label

Delayed Ejaculation

  • Marked delay or inability to ejaculate despite adequate stimulation and desire to climax
  • Medication-induced cases common—SSRIs, antipsychotics, and opioids frequently cause this as a side effect
  • Idiosyncratic masturbation patterns may contribute when partnered stimulation differs significantly from self-stimulation habits

Female Orgasmic Disorder

  • Marked delay, infrequency, or absence of orgasm or significantly reduced orgasmic intensity
  • Situational vs. generalized distinction matters—some women experience orgasm through masturbation but not partnered sex, indicating psychological or relational factors
  • Education often central to treatment—many cases involve insufficient clitoral stimulation or unrealistic expectations based on media portrayals

Compare: Premature Ejaculation vs. Delayed Ejaculation—both are male orgasm-timing disorders, but they represent opposite ends of the spectrum. Premature involves too rapid a response (often serotonin-related), while delayed involves inhibited response (often medication or psychological factors). Essay questions may ask you to explain how the same neurotransmitter system can produce opposite dysfunctions.


Pain disorders create barriers to sexual activity through physical discomfort rather than problems with desire, arousal, or orgasm phases. These conditions often involve both physiological mechanisms (muscle tension, inflammation) and psychological components (fear-avoidance cycles).

Genito-Pelvic Pain/Penetration Disorder

  • Persistent difficulty with vaginal penetration accompanied by pain, fear of pain, or pelvic floor muscle tension
  • Replaces older diagnoses of vaginismus (muscle spasm) and dyspareunia (painful intercourse) in DSM-5, recognizing their overlap
  • Pelvic floor dysfunction central to treatment—physical therapy targeting muscle relaxation, combined with cognitive-behavioral approaches for anxiety and gradual desensitization with dilators

Compare: Genito-Pelvic Pain/Penetration Disorder vs. Female Orgasmic Disorder—both affect women's sexual satisfaction, but pain disorder creates a barrier to sexual activity itself, while orgasmic disorder affects completion of the response cycle. Pain disorders often require medical intervention alongside psychological treatment.


Atypical Arousal Patterns

These conditions involve sexual arousal to unusual stimuli and are only considered disorders when they cause distress or harm. The distinction between paraphilias (atypical interests) and paraphilic disorders (causing distress/harm) is crucial for understanding this category.

Paraphilic Disorders

  • Intense, persistent sexual arousal to atypical stimuli—objects, situations, or non-consenting individuals; includes fetishistic, voyeuristic, and exhibitionistic patterns
  • Disorder requires distress or harm—a paraphilia alone is not pathological; it becomes a disorder when it causes personal distress or involves non-consenting others
  • Treatment varies by type—cognitive-behavioral therapy addresses thought patterns; antiandrogen medications may reduce intensity of urges in severe cases

Compare: Paraphilic Disorders vs. other sexual dysfunctions—most dysfunctions involve problems with the normal sexual response, while paraphilic disorders involve atypical targets of arousal. This is a fundamentally different category that raises distinct ethical and legal considerations.


Quick Reference Table

ConceptBest Examples
Desire-phase disordersFemale Sexual Interest/Arousal Disorder, Male Hypoactive Sexual Desire Disorder, Sexual Aversion Disorder
Arousal-phase disordersErectile Dysfunction, PGAD
Orgasm-phase disordersPremature Ejaculation, Delayed Ejaculation, Female Orgasmic Disorder
Pain-related disordersGenito-Pelvic Pain/Penetration Disorder
Atypical arousal patternsParaphilic Disorders
Primarily physiological etiologyErectile Dysfunction, PGAD, medication-induced Delayed Ejaculation
Primarily psychological etiologySexual Aversion Disorder, performance anxiety-related ED
Biopsychosocial complexityFemale Sexual Interest/Arousal Disorder, Genito-Pelvic Pain/Penetration Disorder

Self-Check Questions

  1. Which two dysfunctions represent opposite problems with orgasm timing in males, and what neurotransmitter system is implicated in both?

  2. How does the DSM-5 distinction between "paraphilia" and "paraphilic disorder" reflect broader principles about when sexual variation becomes pathology?

  3. Compare Sexual Aversion Disorder and Male Hypoactive Sexual Desire Disorder—what underlying mechanisms differentiate active avoidance from low motivation?

  4. A patient reports adequate desire and arousal but cannot reach orgasm during partnered sex, though she can during masturbation. Which disorder does this suggest, and what does the situational pattern indicate about likely causes?

  5. Explain why Genito-Pelvic Pain/Penetration Disorder exemplifies the biopsychosocial model—what biological, psychological, and social factors might interact in this condition?