Dina E. K. H. I. Peters

Definition

Dina E. K. H. I. Peters is a researcher known for her contributions to understanding epigenetic alterations and their implications in toxicology and environmental health. Her work emphasizes how exposure to various environmental agents can lead to changes in gene expression that do not involve alterations to the DNA sequence itself, highlighting the significance of epigenetic mechanisms in the context of disease and development.

5 Must Know Facts For Your Next Test

1. Peters' research illustrates how environmental toxins can induce epigenetic changes that may contribute to diseases like cancer and neurodegenerative disorders.
2. She emphasizes the importance of understanding both genetic and epigenetic factors when assessing individual susceptibility to toxicants.
3. Peters has highlighted the role of dietary components in modulating epigenetic marks, suggesting potential strategies for disease prevention.
4. Her findings stress that epigenetic changes can be reversible, offering potential avenues for therapeutic interventions in affected individuals.
5. Peters' work underlines the complex interplay between genetic predisposition and environmental exposures, contributing significantly to the field of environmental toxicology.

Review Questions

• How does Dina E. K. H. I. Peters' research connect epigenetics with environmental health risks?
  • Dina E. K. H. I. Peters' research highlights the relationship between environmental exposures and epigenetic alterations, showing that toxins can induce changes in gene expression without altering DNA sequences. This work is crucial in understanding how everyday chemicals and pollutants may contribute to diseases, as it suggests that environmental factors can modify our genetic expression over time, increasing susceptibility to various health conditions.
• What are some mechanisms by which epigenetic changes induced by toxins can lead to disease development, according to Peters’ findings?
  • Peters identifies several mechanisms through which toxins induce epigenetic changes, such as DNA methylation and histone modifications, which alter gene expression patterns associated with cell proliferation, differentiation, and apoptosis. These changes can disrupt normal cellular functions and lead to the onset of diseases like cancer or metabolic disorders, demonstrating the critical link between environmental exposures and health outcomes.
• Evaluate the implications of Dina E. K. H. I. Peters’ work on future research and public health strategies regarding toxic exposure.
  • Dina E. K. H. I. Peters’ research has significant implications for future studies and public health strategies by underscoring the need for a comprehensive approach that includes both genetic and epigenetic assessments in risk evaluations for toxic exposure. Her findings advocate for preventive measures that consider dietary influences and lifestyle choices in mitigating disease risk associated with environmental toxins, paving the way for new intervention strategies aimed at reversing harmful epigenetic changes.

Related terms

Epigenetics:

The study of heritable changes in gene expression that do not involve changes to the underlying DNA sequence, often influenced by environmental factors.

DNA Methylation: A key epigenetic mechanism involving the addition of a methyl group to DNA, often affecting gene expression and playing a role in various diseases.

Histone Modification: Chemical alterations to histone proteins that help package DNA, influencing gene accessibility and expression as part of the epigenetic regulation process.